Skip to main navigation
Skip to search
Skip to main content
Princeton University Home
Help & FAQ
Link opens in a new tab
Search content at Princeton University
Home
Profiles
Research units
Facilities
Projects
Research output
Press/Media
Infection-induced lysine lactylation enables herpesvirus immune evasion
Matthew D. Tyl
, Victoria U. Merengwa
,
Ileana M. Cristea
Molecular Biology
Lewis-Sigler Institute for Integrative Genomics
Research output
:
Contribution to journal
›
Article
›
peer-review
17
Link opens in a new tab
Scopus citations
Overview
Fingerprint
Fingerprint
Dive into the research topics of 'Infection-induced lysine lactylation enables herpesvirus immune evasion'. Together they form a unique fingerprint.
Sort by
Weight
Alphabetically
Keyphrases
Lactate
100%
Herpesvirus
100%
Immune Evasion
100%
Lactylation
100%
Lysine Lactylation
100%
Interferon-inducible Protein 16
75%
Immune Signaling
50%
Viral Infection
50%
Virus
25%
Human Cytomegalovirus
25%
Virus Infection
25%
Signal Transduction
25%
Herpes Simplex Virus Type 1 (HSV-1)
25%
Virus Gene
25%
Proviral DNA
25%
Viral Proteins
25%
RNA-binding Protein
25%
Cytomegalovirus Infection
25%
Gene Repression
25%
Regulatory Role
25%
Immune Factors
25%
Viral Spread
25%
Direct Inhibitors
25%
DNA Damage Response
25%
Intrinsically Disordered Region
25%
DNA Biosensor
25%
Protein Condensates
25%
Aerobic Glycolysis
25%
Cytokine Induction
25%
DNA-dependent Protein Kinase (DNA-PK)
25%
Immunology and Microbiology
interferon
100%
Herpesviridae
100%
Viral Disease
100%
Immune Evasion
100%
Lysine
100%
Human Cytomegalovirus
66%
Dynamics
33%
Viral Protein
33%
Virus Gene
33%
Human Alphaherpesvirus 1
33%
Cytomegalovirus Infection
33%
Gene Repression
33%
Cytokine
33%
Protein RNA Binding
33%
DNA Damage Response
33%
Aerobic Glycolysis
33%
Virus
33%
DNA Virus
33%